OHIRA ShuyaSpecially Appointed Assistant Professor
- Laboratory
- Laboratory of Innate Immunity
- Research Theme
- Elucidating mechanisms of intestinal mucosal immune regulation mediated by Paneth cell α-defensin via intestinal microbiota
- Research Keywords
Mucosal immunity, Innate immunity, Paneth cell, α-defensin, Intestinal microbiota, Systemic immunity, Inter-organ communication, Chronic inflammation, Non-communicable diseases
Overview of Research
Unraveling mechanisms of mucosal immune regulation originating from the small intestine
The intestine, composed of small and large intestines, is a multifunctional organ responsible for digestion, nutrient absorption, immune function, and various other physiological processes. Mucosal immunity in the gut is characterized by its ability to tolerate beneficial commensal microbes and dietary antigens while eliminating pathogens and potentially harmful resident bacteria. Although the intestinal microbiota has been identified as a key environmental factor influencing intestinal mucosal immunity, the mechanisms underlying mucosal immune regulation and its impact on systemic metabolism and immunity in distant organs such as the liver and pancreas remain poorly understood.
To address this, our research focuses on α-defensin, an antimicrobial peptide secreted by Paneth cells, which are uniquely located in the small intestine. We aim to clarify how α-defensin regulates mucosal immunity by selectively shaping the microbial communities in both the small and large intestines, thereby contributing to maintaining host health. Our ultimate goal is to provide new preventive and therapeutic strategies for non-communicable diseases such as diabetes and cancer that are associated with an imbalance of the intestinal microbiota.
Charge
- School of Science:
Biological Science course (Macromolecular Functions), Core Laboratories - Graduate School of Life Science:
Division of Life Science, Transdisciplinary Life Science Course, Functional Cellular Sciences
Representative Publications
Yokoi Y, Nakamura R, Ohira S, Takemi S, Ayabe T, Nakamura K. Potential consequences of phototoxicity on cell function during live imaging of intestinal organoids. PLOS ONE 2024. doi.org/10.1371/journal.pone.0313213.
Nakamura S, Nakamura K, Yokoi Y, Shimizu Y, Ohira S, Hagiwara M, Song Z, Gan L, Aizawa T, Hashimoto D, Teshima T, Ouellette AJ, Ayabe T. Decreased Paneth cell α-defensins promote fibrosis in a choline-deficient L-amino acid-defined high-fat diet-induced mouse model of nonalcoholic steatohepatitis via disrupting intestinal microbiota. Sci Rep 2023. doi.org/10.1038/s41598-023-30997-y.
Ohira S, Yokoi Y, Ayabe T, Nakamura K. Efficient and simple genetic engineering of enteroids using mouse isolated crypts for investigating intestinal functions. Biochem Biophys Res Commun 2022. doi.org/10.1016/j.bbrc.2022.11.008.
Kamioka M, Goto Y, Nakamura K, Yokoi Y, Sugimoto R, Ohira S, Kurashima Y, Umemoto S, Sato S, Kunisawa J, Takahashi Y, Domino SE, Renauld JC, Nakae S, Iwakura Y, Ernst PB, Ayabe T, Kiyono H. Intestinal commensal microbiota and cytokines regulate Fut2 + Paneth cells for gut defense. Proc Natl Acad Sci U S A 2022. doi.org/10.1073/pnas.2115230119.
Suzuki K, Nakamura K, Shimizu Y, Yokoi Y, Hagiwara M, Ohira S, Wang Y, Song Y, Aizawa T, Ayabe T. Decrease of alpha-defensin impairs intestinal metabolite homeostasis via dysbiosis in mouse chronic social defeat stress model. Sci Rep 2021. doi.org/10.1038/s41598-021-89308-y.
Shimizu Y, Nakamura K, Yoshii A, Yokoi Y, Kikuchi M, Shinozaki R, Nakamura S, Ohira S, Sugimoto R, Ayabe T. Paneth cell alpha-defensin misfolding correlates with dysbiosis and ileitis in Crohn’s disease model mice. Life Sci Alliance 2020. doi.org/10.26508/lsa.201900592.
Nakamura K, Yokoi Y, Fukaya R, Ohira S, Shinozaki R, Nishida T, Kikuchi M, Ayabe T. Expression and Localization of Paneth Cells and Their α-Defensins in the Small Intestine of Adult Mouse. Front Immunol 2020. doi.org/10.3389/fimmu.2020.570296.
Refer to HOKKAIDO UNIVERSITY RESEARCHERS DIRECTORY
Note
<Office Hour>
– Time: Anytime during the lecture period
– Place: Frontier-AMLS, 4F
Please contact in advance by E-mail.
E-mail: s.ohira[at]sci.hokudai.ac.jp
Affiliation
- Faculty of Advanced Life Science, Department of Functional Life Sciences, Embryonic and Genetic Engineering
- Frontier Research Center for Advanced Material and Life Science, Academia-Industry Collaboration Unit